Betaine (aka TMG or Trimethylglycine)

A forum to discuss diet and dietary supplements.

Betaine (aka TMG or Trimethylglycine)

Postby Biologist » Mon Aug 31, 2009 10:33 pm

I take a gram of Betaine, also known as TMG or Trimethylglycine, every day and will likely double my dosage of it starting now. Among other things, it's great at knocking down homocysteine. Dr. Kilmer McCully and Dr. Graveline have both discussed the value of methylation, and this molecule is a great source of donor methyl groups for a wide range of important biochemical processes in the body. It is the only supplements that I prefer to take by itself with a meal rather than along with other supplements since it may have a higher probability of reacting with them in the digestive tract. Maybe not, but I take it alone just to be sure. Note that suppression of NF Kappa B (which this substance does) is the primary mechanism by which statins do their work. Statins would be fine if not for their major-league adverse side effects which, ironically, includes the counterproductive reduction of normal and healthy cholesterol levels in most people. This stuff does not have such nasty side effects, it is not hard to find, and is not expensive. If you are not up on interpreting such research abstracts (which is probably good as it means you may "have a life"), skip the rest of this post, but consider taking Betaine.

The following study abstracts were compiled by another "Google researcher" and all comments found in brackets below are his. I am meerly posting his work "as is" starting with the following sentence.


[Mortality in aged humans is associated with reduced muscle strength, and
increased NF Kappa B expression. Betaine has been proven to increase muscle
endurance in young humans, and to decrease NF Kappa B in aged rats. It would be
interesting to see the results of an intervention study of these parameters in
aged humans. It has also been proposed that the relative longevity of bats is
due to their proteins being less vulnerable to urea induced denaturation than
those of mice. Betaine just happens to directly inhibit urea induced

[Dosage of betaine used below was 2.5 gm/day.]

J Int Soc Sports Nutr. 2009 Feb 27;6:7.
Effect of betaine supplementation on power performance and fatigue.
Hoffman JR, Ratamess NA, Kang J, Rashti SL, Faigenbaum AD. Department of
Health and Exercise Science, The College of New Jersey, PO Box 7718, Ewing, New
Jersey 08628, USA.
BACKGROUND: The purpose of this study was to examine the efficacy of
15 days of betaine supplementation on muscle endurance, power performance and
rate of fatigue in active college-aged men.METHODS: Twenty-four male subjects
were randomly assigned to one of two groups. The first group (BET; 20.4 +/- 1.3
years; height: 176.8 +/- 6.6 cm; body mass: 77.8 +/- 13.4 kg) consumed the
supplement daily, and the second group (PL; 21.4 +/- 4.7 years; height: 181.3
+/- 5.9 cm; body mass: 83.3 +/- 5.2 kg) consumed a placebo. Subjects were tested
prior to the onset of supplementation (T1) and 7 (T2) and 14 days (T3) following
supplementation. Each testing period occurred over a 2-day period. During day
one of testing subjects performed a vertical jump power (VJP) and a bench press
throw (BPT) power test. In addition, subjects were required to perform as many
repetitions as possible with 75% of their 1-RM in both the squat and bench press
exercises. Both peak and mean power was assessed on each repetition. On day two
of testing subjects performed two 30-sec Wingate anaerobic power tests (WAnT),
each test separated by a 5-min active rest. RESULTS: No differences were seen at
T2 or T3 in the repetitions performed to exhaustion or in the number of
repetitions performed at 90% of both peak and mean power between the groups in
the bench press exercise. The number of repetitions performed in the squat
exercise for BET was significantly greater (p < 0.05) than that seen for PL at
T2. The number of repetitions performed at 90% or greater of peak power in the
squat exercise was significantly greater for BET at both T2 and T3 than PL. No
differences in any power assessment (VJP, BPT, WAnT) was seen between the groups.
CONCLUSION: Two-weeks of betaine supplementation in active, college males
appeared to improve muscle endurance of the squat exercise, and increase the
quality of repetitions performed.
PMID: 19250531

Free text:

[High doses of betaine returned nfkappab to youthful levels.]

Biol Pharm Bull. 2007 Dec;30(12):2244-9.
Betaine modulates age-related NF-kappaB by thiol-enhancing action.
Go EK, Jung KJ, Kim JM, Lim H, Lim HK, Yu BP, Chung HY. Department of
Pharmacy, College of Pharmacy, Pusan National University, Gumjung-ku, Busan
609-735, Korea.
Depletion of glutathione levels and perturbations in redox status are
considered to play a crucial role in aging and chronic inflammatory processes
through the activation of redox sensitive transcription factors, including
nuclear factor-kappaB (NF-kappaB). In the current study, we assessed the
regulatory action of dietary betaine in the suppression of NF-kappaB by
comparing kidney tissue from old, betaine-supplemented rats or
non-betaine-supplemented rats (age 21 months) and 7 month-old rats. In addition,
cultured HEK 293T cells were utilized for the molecular assessment of betaine's
restorative ability of redox status when treating cells with potent glutathione
(GSH)-depleting agents. Results showed that in old rats a short-term feeding (10
d) with betaine attenuated the age-related decrease in thiol levels, increase in
reactive species and TNFalpha expression via NF-kappaB activation, compared to
the young controls. These findings were verified in the cell-cultured system.
Further investigations found that redox imbalance due to thiol depletion caused
increased NF-kappaB activation, and cyclooxygenase (COX)-2 and TNFalpha levels,
both of which were suppressed by betaine treatment. Based on both in vivo and in
vitro data, we concluded that betaine exerts its efficacy by maintaining thiol
status in the regulation of COX-2 and TNFalpha via NF-kappaB activation during
PMID: 18057706

Free text:

[If protein denaturation is a major driving force behind aging, then betaine
could be expected to slow this process.]

J Phys Chem B. 2009 Apr 16;113(15):5327-38.
Osmolyte counteracts urea-induced denaturation of alpha-chymotrypsin.
Venkatesu P, Lee MJ, Lin HM. Department of Chemical Engineering, National
Taiwan University of Science and Technology, 43 Keelung Road, Section 4, Taipei
106-07, Taiwan.
The stability of proteins is reduced by urea, which is methylamine and
nonprotecting osmolyte; eventually urea destabilizes the activity and function
and alters the structure of proteins, whereas the stability of proteins is
raised by the osmolytes, which are not interfering with the functional activity
of proteins. The deleterious effect of urea on proteins has been counteracted by
methylamines (osmolytes), such as trimethylamine N-oxide (TMAO), betaine, and
sarcosine. To distinctly enunciate the comparison of the counteracting effects
between these methylamines on urea-induced denaturation of alpha-chymotrypsin
(CT), we measured the hydrodynamic diameter (d(H)) and the thermodynamic
properties (T(m), DeltaH, DeltaG(U), and DeltaC(p)) with dynamic light
scattering (DLS) and differential scanning calorimeter (DSC), respectively. The
present investigation compares the compatibility and counteracting hypothesis by
determining the effects of methylamines and urea, as individual components and
in combination at a concentration ratio of 1:2 (methylamine:urea) as well as
various urea concentrations (0.5-5 M) in the presence of 1 M methylamine. The
experimental results revealed that the naturally occurring osmolytes TMAO,
betaine, and sarcosine strongly counteracted the urea actions on
alpha-chymotrypsin. The results also indicated that TMAO counteracting the urea
effects on CT was much stronger than betaine or sarcosine.
PMID: 19354310

Am J Clin Nutr. 2008 Feb;87(2):424-30.
Comment in:
Am J Clin Nutr. 2008 Feb;87(2):277-8.
Am J Clin Nutr. 2008 Jul;88(1):247-8; author reply 248.
Dietary choline and betaine intakes in relation to concentrations of
inflammatory markers in healthy adults: the ATTICA study.
Detopoulou P, Panagiotakos DB, Antonopoulou S, Pitsavos C, Stefanadis C.
Department of Nutrition Science-Dietetics, Harokopio University, Athens, Greece.
BACKGROUND: Choline and betaine are found in a variety of plant and animal
foods and were recently shown to be associated with decreased homocysteine
concentrations. OBJECTIVE: The scope of this work was to investigate the
associations between dietary choline and betaine consumption and various markers
of low-grade systemic inflammation. DESIGN: Under the context of a
cross-sectional survey that enrolled 1514 men (18-87 y of age) and 1528 women
(18-89 y of age) with no history of cardiovascular disease (the ATTICA Study),
fasting blood samples were collected and inflammatory markers were measured.
Dietary habits were evaluated with a validated food-frequency questionnaire, and
the intakes of choline and betaine were calculated from food-composition tables.
RESULTS: Compared with the lowest tertile of choline intake (<250 mg/d),
participants who consumed >310 mg/d had, on average, 22% lower concentrations of
C-reactive protein (P < 0.05), 26% lower concentrations of interleukin-6 (P <
0.05), and 6% lower concentrations of tumor necrosis factor-alpha (P < 0.01).
Similarly, participants who consumed >360 mg/d of betaine had, on average, 10%
lower concentrations of homocysteine (P < 0.01), 19% lower concentrations of
C-reactive protein (P < 0.1), and 12% lower concentrations of tumor necrosis
factor-alpha (P < 0.05) than did those who consumed <260 mg/d. These findings
were independent of various sociodemographic, lifestyle, and clinical
characteristics of the participants. CONCLUSIONS: Our results support an
association between choline and betaine intakes and the inflammation process in
free-eating and apparently healthy adults. However, further studies are needed
to confirm or refute our findings.
PMID: 18258634 [PubMed

Free text:

Br J Nutr. 2007 Nov;98(5):960-8. Epub 2007 May 31.
The association of betaine, homocysteine and related metabolites with cognitive
function in Dutch elderly people.
Eussen SJ, Ueland PM, Clarke R, Blom HJ, Hoefnagels WH, van Staveren WA, de
Groot LC. Division of Human Nutrition, Wageningen University, P.O. Box 8129 6700
EV Wageningen, The Netherlands.
The importance of the one-carbon metabolites, choline and homocysteine, to
brain function is well known. However, the associations between the one-carbon
metabolites choline, betaine, methionine and dimethylglycine with cognition in
elderly are unclear. We therefore examined the associations of these metabolites
with cognition in a double-blind, placebo-controlled trial. Individuals (n 195)
were randomized to receive daily oral capsules with either 1000 microg cobalamin
(vitamin B12), or 1000 microg cobalamin plus 400 microg folic acid, or placebo
for 24 weeks. Concentrations of homocysteine, methionine, choline, betaine and
dimethylglycine were assessed before and after 12 and 24 weeks of treatment.
Cognitive function, including domains of attention, construction, sensomotor
speed, memory and executive function, was assessed before and after 24 weeks of
treatment. At baseline, elevated plasma homocysteine was associated with lower
performance of attention, construction, sensomotor speed and executive function.
In addition, betaine was positively associated with better performance of
construction, sensomotor speed and executive function, whereas elevated
concentrations of methionine were positively associated with sensomotor speed.
Daily combined supplementation with cobalamin plus folic acid decreased total
homocysteine concentrations by 36%, and increased betaine concentrations by 38%.
Participants with the largest increases in betaine concentrations showed a
borderline significant (P = 0.07) higher memory performance compared to those
without it. Although this trial observed associations of homocysteine and
betaine with cognitive domains prior to supplementation, decreased
concentrations of homocysteine were not related to improved cognitive
performance. There was a tendency of participants with the largest increases in
betaine concentrations to show the greatest improvement in memory function.
PMID: 17537289

Free text:

J Radiat Res (Tokyo). 2005 Mar;46(1):117-21.
Glycine betaine, a beer component, protects radiation-induced injury.
Monobe M, Uzawa A, Hino M, Ando K, Kojima S. Department of Radiation
Biosciences, Faculty of Pharmaceutical Sciences, Tokyo University of Science,
Noda-shi, Chiba, Japan.
Human whole-blood was exposed to 137Cs gamma-rays or 50 keV/microm carbon
ions in the presence or absence of glycine betaine, a beer component in vitro.
The dicentrics of chromosome aberrations in human lymphocytes were significantly
(p < 0.05) reduced by glycine betaine after irradiation with 4 Gy of either
gamma-rays or carbon ions. The maximum protection by glycine betaine for
gamma-rays or carbon ions was 37% and 20%, respectively. C3H/He female mice,
aged 14 weeks, received an i.p. injection of glycine betaine 15 min before
whole-body irradiation with gamma-rays or 50 keV/microm carbon ions. Glycine
betaine significantly (p < 0.05) increased the percent survival of irradiated
mice with either gamma-rays or carbon ions. In conclusion, glycine betaine is a
potent protector against damages caused by low- and high-LET radiation.
PMID: 15802867

Free text:

Br J Nutr. 2004 Oct;92(4):665-9.
The effect of low doses of betaine on plasma homocysteine in healthy volunteers.
Alfthan G, Tapani K, Nissinen K, Saarela J, Aro A. Department of Health and
Functional Capacity, National Public Health Institute (KTL), Helsinki, Finland.
Homocysteine is a risk factor for vascular diseases, and lowering of plasma
total homocysteine (tHcy) may be beneficial for health. Homocysteine can be
remethylated to methionine by betaine-homocysteine methyltransferase using
betaine (2(N,N,N-trimethyl)glycine) as methyl donor. A dose of 6 g betaine/d has
been used in the treatment of homocystinuria, but data on the dose-response are
scarce. Thirty-four healthy men and women were supplied with doses of 1, 3 and 6
g betaine and then with 6 g betaine+1 mg folic acid for four consecutive 1-week
periods. The mean plasma tHcy concentration decreased by 1.1 (NS), 10.0 and 14.0
% (P<0.001) after supplementation with 1, 3 and 6 g betaine respectively. A
further decrease in plasma tHcy by 5 % (P<0.01) was achieved by combining 1 mg
folic acid with the 6 g betaine dose. Plasma betaine increased from 31 (sd 13)
to 255 (sd 136) mumol/l in a dose-dependent manner (R(2) 0.97). We conclude that
plasma tHcy is lowered rapidly and significantly by 3 or 6 g betaine/d in
healthy men and women.
PMID: 15522136

Free text:

Am J Gastroenterol. 2001 Sep;96(9):2711-7.
Comment in:
Am J Gastroenterol. 2001 Sep;96(9):2534-6.
Betaine, a promising new agent for patients with nonalcoholic steatohepatitis:
results of a pilot study.
Abdelmalek MF, Angulo P, Jorgensen RA, Sylvestre PB, Lindor KD. Divisions of
Gastroenterology and Hepatology and Surgical Pathology, Mayo Clinic and
Foundation, Rochester, Minnesota 55905, USA.
OBJECTIVES: No effective therapy currently exists for patients with
nonalcoholic steatohepatitis (NASH). Betaine, a naturally occurring metabolite
of choline, has been shown to raise S-adenosylmethionine (SAM) levels that may
in turn play a role in decreasing hepatic steatosis. Our aim was to determine
the safety and effects of betaine on liver biochemistries and histological
markers of disease activity in patients with NASH. METHODS: Ten adult patients
with NASH were enrolled. Patients received betaine anhydrous for oral solution
(Cystadane) in two divided doses daily for 12 months. Seven out of 10 patients
completed 1 yr of treatment with betaine. RESULTS: A significant improvement in
serum levels of aspartate aminotransferase (p = 0.02) and ALAT (p = 0.007)
occurred during treatment. Aminotransferases normalized in three of seven
patients, decreased by >50% in three of seven patients, and remained unchanged
in one patient when compared to baseline values. A marked improvement in serum
levels of aminotransferases (ALT -39%; AST -38%) also occurred during treatment
in those patients who did not complete 1 yr of treatment. Similarly, a marked
improvement in the degree of steatosis, necroinflammatory grade, and stage of
fibrosis was noted at 1 yr of treatment with betaine. Transitory GI adverse
events that did not require any dose reduction or discontinuation of betaine
occurred in four patients. CONCLUSIONS: Betaine is a safe and well tolerated
drug that leads to a significant biochemical and histological improvement in
patients with NASH. This novel agent deserves further evaluation in a
randomized, placebo-controlled trial.
PMID: 11569700

Acta Odontol Scand. 1998 Apr;56(2):65-9.
Betaine-containing toothpaste relieves subjective symptoms of dry mouth.
Soderling E, Le Bell A, Kirstila V, Tenovuo J. Institute of Dentistry,
University of Turku, Finland.
Subjects with dry mouth often experience irritation of the oral mucosa when
using sodium lauryl sulfate containing products for oral hygiene. Betaine, or
trimethylglycine, reduces skin-irritating effects of ingredients of cosmetics
such as sodium lauryl sulfate. The aim of the present study was to compare the
effects of a betaine-containing toothpaste with a regular toothpaste on the oral
microbial flora, the condition of the oral mucosa, and subjective symptoms of
dry mouth in subjects with chronic dry mouth symptoms. Thirteen subjects with
chronic dry mouth symptoms and with a paraffin-stimulated salivary flow rate <
or = 1 mL/min participated in the double-blind crossover study. Ten subjects had
a very low salivary flow rate (< or = 0.6 mL/min). The subjects used both
experimental toothpastes (with or without 4% betaine) twice a day for 2 weeks.
Oral examinations and microbiologic sample collections were made at the base
lines preceding the two experimental periods and at the end. Standardized
questions on subjective symptoms of dry mouth were used when the subjects were
interviewed at the end of the two experimental periods. No study-induced
significant changes were observed in the microbiologic variables (plaque index,
mutans streptococci, lactobacilli, Candida species) or in the appearance of the
oral mucosa. The use of the betaine-containing toothpaste was, however,
associated with a significant relief of several subjective symptoms of dry
mouth. Betaine appears thus to be a promising ingredient of toothpastes in
general and especially of toothpastes designed for patients with dry mouth.
PMID: 9669455
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Postby David Staup » Tue Sep 01, 2009 1:01 am


would you list the suppliments and doses that you take.
I'd like to see if there are any more potential improvements that I've missed in this mass of information.
David Staup
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Postby cjbrooksjc » Tue Sep 01, 2009 5:03 am

B: From Wikipedia:

[Quote] NF-κB plays a key role in regulating the immune response to infection. Conversely, incorrect regulation of NF-κB has been linked to cancer, inflammatory and autoimmune diseases, septic shock, viral infection, and improper immune development. NF-κB has also been implicated in processes of synaptic plasticity and memory.[6] [Quote]

Any concerns there? I already am taking a steroid and would be reticent to compound that effect, and my memory is bad enough as is. :wink:

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Postby Biologist » Wed Sep 02, 2009 10:45 am

Hi, David and Brooks.

I have ideas regarding both of your posts but am covered up in "time sensitive" projects right now (i.e., "dead lines"), and will be at least for the next several days. Hold your thoughts. I'll be back.

Hey, Brooks, think Vitamin D. It has been the missing regulator out of the mix for the last decade or two; not to mention cholesterol, the great inflamation fighter/fixer.

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Postby cjbrooksjc » Wed Sep 02, 2009 11:11 am

Biologist: Thanks for the advice. I take a boatload of VitD as a result of earlier (yours included) exchanges on the subj. My hair and nails grow so fast I'm thinking of becoming a barber - I spend so much time in the shop.

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Postby David Staup » Wed Sep 02, 2009 12:18 pm

Thanks for responding, no hurry I'm cautious and only add one new thing at a time anyway and will want several weeks on the TMG before I can evaluate its effects and I won't even start that until I see what the high doses of magnesium oratate do when and if I get to the high dose level if I even can (had adverse reaction by starting at 1500mg)

There are some benifits to being bald after all :wink:

David Staup
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Postby Biologist » Wed Sep 02, 2009 1:37 pm

David, I'm right with you on the addition of new supplements thing. When you get around to TMG, consider getting your homosysteine levels checked before doing the supplementing as a baseline. Otherwise, I am unconvinced we will be able to detect the good it may be doing because of the ebbs and flow of our condition, as you cited previously.

If you want a supplement that you can feel, try GABA. It is a simple amino acid like the 20 or so that make up our proteins, but this one is different. It is not a constituent of proteins, but rather acts as a neurotransmitter. An inhibitory one. I use it as a sleep aid. I can definitely detect it about half an hour after taking it on an empty stomach. Even gives "a taste sensation" in the mouth, at least for me, after about 30 minutes or so. My HRT doctor mentioned it, and uses it himself, as a sleep aid. I take 750 mg at night before bed. If you do not feel it, try doubling the dosage.

(While I'm thinking of it, D-Ribose may be something to try as a "sleep aid" for those with sleep apnea; it may strenghen tonic muscle tone for keeping air ways unobstructed -- and Vinpocetine by reducing the number of disruption cycles by increasing brain oxygenation -- just some thoughts. My sleeping has improved in recent months.)

I am reading about GABA's mechanism of action now in Chapter 45 "Organization of the Nervous System; Basic Functions of Synapses and Transmitter Substances" from my "Text Book of Medical Physiology" book by Guyton & Hall. (These two guys will certainly go down in medical history, particularly Guyton, but Hall is still "up and coming"). Fascinating!


OK, nice break, but back to work for me...

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Postby Biologist » Wed Sep 02, 2009 6:34 pm

Quick note:

If you have ALS-like symptoms, go slow on the GABA. It will make symptoms somewhat more pronounced until it wears off after several hours.

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Postby David Staup » Thu Sep 03, 2009 11:29 am

"David, I'm right with you on the addition of new supplements thing. When you get around to TMG, consider getting your homosysteine levels checked before doing the supplementing as a baseline. Otherwise, I am unconvinced we will be able to detect the good it may be doing because of the ebbs and flow of our condition, as you cited previously."

alas since the beginning of this year I have no insurance and the one doctor I trust charges $75.00 per visit. this is fair because he will spend up to an hour with me and is honorable enough to look at my assertions objectively...adding the cost of tests is a bit much for me as I live on social security disability and do not yet qualify for health care.

I am more interested in mitigating my symptoms from statin damage first as the kidney problems probably contributed to high homocysteine. since reducing my activities and using the electrolyte solution my kidney function has improved and I currently suppliment with folic acid (400mcg x2/day) plus b6 and b12 which should help. Do you think I would need to add the TMG?

As for the GABA I have no problem going to sleep just wake after 2-4 hours and need to loosen up before going back to sleep. this same thing happens during the day if I don't walk some every 1-2 hours.

David Staup
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Postby Biologist » Fri Sep 04, 2009 10:38 am

Hi, David.

I tried to find an older post of mine (with search terms I know for sure are in the text) that would have been appliclable here as a response. It appears the Search Function cuts off searching older posts at some point back in time. That is REAL unfortunate. The website, ironically, has impaired memory! I've run into this problem before here. May be a limitation of the softwear, but if it's meerly a setting that can be changed, the Administrator may want to consider resetting it such that older posts may be found and retrieved.

The reason for my bringing up homosysteine testing in my previous post was only as an available means for detecting effacy for the TMG supplementation. For many of the supplements we take, we cannot expect to be able to detect that they do good things for us -- or know if they are doing nothing, or even harm. I do not think you need to do such testing to take TMG. However, with your folic acid, B6 and B12, you should be in good shape on the homosysteine department without it.

I have not gotten around yet to some of Dr. Gravelines suggestions in his most recent book. But I'm glad their there. When the time is right, I know where to find them.

I think you are on the right tract in getting your statin issues resolved before doing other things that may or may not be cost effective for general health. The draining of resources is one of the most damnable things about the pharmaceutical parasite. The medical profession has insisted in recent years that convicted felons (such as Pfizer, as reverified by their most recent conviction earlier this week) are the best source of medical advice to be applied to their patients. A bizarre concept, if you ask me. However, our doctors must make this decision. If doctor's insist, for instance, that the nation's arsonists and/or embezzlers have all the best medical answers, while I find that hard to believe, its up to them -- its their call. However, if the "criminal of choice" happens to be recidivist poisoners, and it adversly effects me and my family, I have to do more than seriously question their judgment, medical or otherwise. I have to pass on the costs. Doctors need to be incentivized to make better decisions about where by get their medical advice, it seems to me. If they start footing the bill for some the damage caused by questionable judgment, as some are getting the opportunity to do -- including my own former doctor, we may start seeing some improved medical outcomes -- and less draining of resources.

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Postby Biologist » Fri Sep 04, 2009 10:44 am

Typos corrected for last sentence:

"Doctors need to be incentivized to make better decisions
about where they get their medical advice, it seems to me.
If they start footing the bill for some of the damage caused
by their questionable judgment, as some are getting the
opportunity to do -- including my own former doctor, we
may start seeing some improved medical outcomes --
and less draining of resources. "

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Postby David Staup » Fri Sep 04, 2009 2:28 pm


"If they start footing the bill for some of the damage caused
by their questionable judgment, as some are getting the
opportunity to do -- including my own former doctor"

very interesting statement I wish you luck. keep us informed!

unfortunately I live in Texas where they passed tort reform several years ago and the lawyers just laugh at any suggestion of a lawsuit for this.
My only hope is a federal investigation into the MD side of the picture and that's not likely to happen.

David Staup
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Postby Brian C. » Sat Sep 05, 2009 1:20 am

I came to the conclusion long ago that basically the World is run by criminals for the benefit of criminals.

Brian C.
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statin drugs

Postby gotts1936 » Sat Sep 05, 2009 9:58 am

I have come to the conclusion the all medication (drugs) are toxic to some individuals, therefore, until test are designed to determine if a particular individual will have a bad reaction to a recommended medication, I believe that individual should not take it.

Until we stop accepting the medical profession from using us as guines pigs nothing will change. May be it is time for political action. I am not talking about marching down the street, but what would happen if everyone stopped buying their medication? Just a thought, but that sure would wake up those who think we are all idiots!
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Postby Brian C. » Sat Sep 05, 2009 12:50 pm

The first step is recalibrate one's perception of doctors from demi-god status to that of just another tradesperson. Engage them to do a particular job for you then pay them off after (and only after) satisfactory completion. Perhaps a "due diligence" clause should be introduced into a contractual obligation between the client and the practitioner. If the practitioner can be shown to have not exercised such in regard to any treatment prescribed then a financial penalty shall be incurred.

Hah! Fat chance.

Brian C.
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statin drugs

Postby gotts1936 » Sat Sep 05, 2009 1:22 pm

If America had the Marxist healthcare system you do in the UK, I could agree with you, but here in America our doctors are controlled by the drug companies and the artificial symptom levels they set that require our doctors to prescribe toxic medications to their patients or they could be charged with malpractice.

Also, many doctors due not report the medication side effects or complications that occur with medications they have prescribed. If they do, the drug companies and federal government make their life HELL!

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Postby Biologist » Sat Sep 12, 2009 1:53 pm

I am still too busy to be doing much posting for a while, but I am going to do this one anyway to correct an error of mine regarding finding old posts. The following hyperlink is the post that I could not find earlier. It appears that I was the one who was memory impaired as I had forgotten about the site's Google function that was added in recent months. It makes finding old posts very easy.

David, if you read the following hyperlinked post you will see that the drug companies are not nearly so heartless as they may at first appear. They actually provide a suggestion for attaining the necessary resources for purchasing their drugs, if you look carefully. :evil:

Brian's hyperlink from the first post in the above thread is worth a re-read for all women. Note that Cornell Law School’s Ted Eisenberg, who is cited in the article, makes "an appearance" in the subsequent comments section of the webpage to make short work of the reply from Pfizer's friendly spokesperson. (I suspect such lawsuits, specificlly regarding women and statins, are not unlikely in the future to recover state moneys as state economies falter further. The political dynamics and loyalties may shift for state politicians, including state prosecutors from AG's offices. It's a decent way to become state Governor.)

And right before that comment from Ted was this one from Laurie:

“'they were not designed to specifically study women.' â€Â
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Postby Biologist » Sat Sep 12, 2009 1:54 pm

This was intended to be my first hyperlink above:

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Postby rkcannon » Sun Jul 25, 2010 11:41 pm

Great ideas- everyone stop buying medication, and require "due diligence" by their doctors. We are really being duped. It's amazing the brainwashing they've done to an entire society.

My question is about methylation and mercury. I really think part of the problem with statins is they open up the cells to heavy metals to become toxic which further causes problems. It snowballs. Then it is very difficult to get rid of the mercury especially. Also NAC N Aceytl Cysteine is supposed to help glutathione formation, which helps reduce the cellular toxicity. Seems like this would be an important supplement.
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